Publikationen (12) Publikationen von Ester Gangoso Rodríguez

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2023

  1. Elevated FOXG1 in glioblastoma stem cells cooperates with Wnt/β-catenin to induce exit from quiescence

    Cell Reports, Vol. 42, Núm. 6

2022

  1. Lrig1 regulates the balance between proliferation and quiescence in glioblastoma stem cells

    Frontiers in Cell and Developmental Biology, Vol. 10

2021

  1. Glioblastomas acquire myeloid-affiliated transcriptional programs via epigenetic immunoediting to elicit immune evasion

    Cell, Vol. 184, Núm. 9, pp. 2454-2470.e26

2018

  1. Modelling glioblastoma tumour-host cell interactions using adult brain organotypic slice co-culture

    Disease models & mechanisms, Vol. 11, Núm. 2

  2. Modelling glioblastoma tumour-host cell interactions using adult brain organotypic slice co-culture

    DMM Disease Models and Mechanisms, Vol. 11, Núm. 2

2017

  1. A c-Src inhibitor peptide based on connexin43 exerts neuroprotective effects through the inhibition of glial hemichannel activity

    Frontiers in Molecular Neuroscience, Vol. 10

  2. Efficient CRISPR/cas9-assisted gene targeting enables rapid and precise genetic manipulation of mammalian neural stem cells

    Development (Cambridge), Vol. 144, Núm. 4, pp. 635-648

  3. Elevated FOXG1 and SOX2 in glioblastoma enforces neural stem cell identity through transcriptional control of cell cycle and epigenetic regulators

    Genes and Development, Vol. 31, Núm. 8, pp. 757-773

2016

  1. General anesthetics have differential inhibitory effects on gap junction channels and hemichannels in astrocytes and neurons

    GLIA, Vol. 64, Núm. 4, pp. 524-536

  2. The role of connexin43-Src interaction in astrocytomas: A molecular puzzle

    Neuroscience, Vol. 323, pp. 183-194

2014

  1. A cell-penetrating peptide based on the interaction between c-Src and connexin43 reverses glioma stem cell phenotype.

    Cell death & disease, Vol. 5

2012

  1. Reduced connexin43 expression correlates with c-Src activation, proliferation, and glucose uptake in reactive astrocytes after an excitotoxic insult

    GLIA, Vol. 60, Núm. 12, pp. 2040-2049