INSTITUTO DE BIOLOGÍA FUNCIONAL Y GENÓMICA
Instituto
Institut d'Investigació Biomédica de Bellvitge
Barcelona, EspañaPublicaciones en colaboración con investigadores/as de Institut d'Investigació Biomédica de Bellvitge (12)
2021
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Disruption of NIPBL/Scc2 in Cornelia de Lange Syndrome provokes cohesin genome-wide redistribution with an impact in the transcriptome
Nature Communications, Vol. 12, Núm. 1
2019
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Cdc14 activation requires coordinated Cdk1-dependent phosphorylation of Net1 and PP2A–Cdc55 at anaphase onset
Cellular and Molecular Life Sciences, Vol. 76, Núm. 18, pp. 3601-3620
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c-Met Signaling Is Essential for Mouse Adult Liver Progenitor Cells Expansion After Transforming Growth Factor-β-Induced Epithelial–Mesenchymal Transition and Regulates Cell Phenotypic Switch
Stem Cells, Vol. 37, Núm. 8, pp. 1108-1118
2018
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Bone morphogenetic protein 9 as a key regulator of liver progenitor cells in DDC-induced cholestatic liver injury
Liver International, Vol. 38, Núm. 9, pp. 1664-1675
2017
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ORP-Mediated ER Contact with Endocytic Sites Facilitates Actin Polymerization
Developmental Cell, Vol. 43, Núm. 5, pp. 588-602.e6
2016
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Dissecting the role of epidermal growth factor receptor catalytic activity during liver regeneration and hepatocarcinogenesis
Hepatology, Vol. 63, Núm. 2, pp. 604-619
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Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356
Autophagy
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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Autophagy, Vol. 12, Núm. 1, pp. 1-222
2015
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HGF/c-Met signaling promotes liver progenitor cell migration and invasion by an epithelial-mesenchymal transition-independent, phosphatidyl inositol-3 kinase-dependent pathway in an in vitro model
Biochimica et Biophysica Acta - Molecular Cell Research, Vol. 1853, Núm. 10, pp. 2453-2463
2013
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BMP9 Is a Proliferative and Survival Factor for Human Hepatocellular Carcinoma Cells
PLoS ONE, Vol. 8, Núm. 7
2012
2007
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Activation of NADPH oxidase by transforming growth factor-β in hepatocytes mediates up-regulation of epidermal growth factor receptor ligands through a nuclear factor-κB-dependent mechanism
Biochemical Journal, Vol. 405, Núm. 2, pp. 251-259