Sandra María Sancho Martínez-rekin lankidetzan egindako argitalpenak (7)

2020

  1. Impaired Tubular Reabsorption Is the Main Mechanism Explaining Increases in Urinary NGAL Excretion Following Acute Kidney Injury in Rats

    Toxicological sciences : an official journal of the Society of Toxicology, Vol. 175, Núm. 1, pp. 75-86

  2. Pathophysiological mechanisms underlying a rat model of triple whammy acute kidney injury

    Laboratory Investigation, Vol. 100, Núm. 11, pp. 1455-1464

2016

  1. Mechanisms of triple whammy acute kidney injury

    Pharmacology and Therapeutics, Vol. 167, pp. 132-145

2015

  1. Acute tubular necrosis: An old term in search for a new meaning within the evolving concept of acute kidney injury

    New Horizons in Translational Medicine, Vol. 2, Núm. 4-5, pp. 110-117

  2. Urinary proteomics in renal pathophysiology: Impact of proteinuria

    Proteomics - Clinical Applications, Vol. 9, Núm. 5-6, pp. 636-640

2011

  1. Urinary levels of regenerating islet-derived protein III Β and gelsolin differentiate gentamicin from cisplatin-induced acute kidney injury in rats

    Kidney International, Vol. 79, Núm. 5, pp. 518-528

2010

  1. Sub-nephrotoxic doses of gentamicin predispose animals to developing acute kidney injury and to excrete ganglioside M2 activator protein

    Kidney International, Vol. 78, Núm. 10, pp. 1006-1015