Publications in collaboration with researchers from Instituto de Biología Molecular y Celular del Cáncer de Salamanca / Centro de Investigación del Cáncer (11)

2024

  1. Platelet C3G: a key player in vesicle exocytosis, spreading and clot retraction

    Cellular and Molecular Life Sciences, Vol. 81, Núm. 1

2022

  1. New functions of C3G in platelet biology: Contribution to ischemia-induced angiogenesis, tumor metastasis and TPO clearance

    Frontiers in Cell and Developmental Biology, Vol. 10

2020

  1. Transcriptomic analysis of patients with immune thrombocytopenia treated with eltrombopag

    Platelets, Vol. 31, Núm. 8, pp. 993-1000

2018

  1. C3G, through its GEF activity, induces megakaryocytic differentiation and proplatelet formation

    Cell Communication and Signaling, Vol. 16, Núm. 1

  2. How Rap and its GEFs control liver physiology and cancer development. C3G alterations in human hepatocarcinoma

    HEPATIC ONCOLOGY, Vol. 5, Núm. 1

2017

  1. C3G promotes a selective release of angiogenic factors from activated mouse platelets to regulate angiogenesis and tumor metastasis

    Oncotarget, Vol. 8, Núm. 67, pp. 110994-111011

  2. The CRISPR/Cas9 system efficiently reverts the tumorigenic ability of BCR/ABL in vitro and in a xenograft model of chronic myeloid leukemia

    Oncotarget, Vol. 8, Núm. 16, pp. 26027-26040

2015

  1. p38 MAPK down-regulates fibulin 3 expression through methylation of gene regulatory sequences: Role in migration and invasion

    Journal of Biological Chemistry, Vol. 290, Núm. 7, pp. 4383-4397

2007

  1. RET/papillary thyroid carcinoma oncogenic signaling through the Rap1 small GTPase

    Cancer Research, Vol. 67, Núm. 1, pp. 381-390

2006

  1. Characterization of p87C3G, a novel, truncated C3G isoform that is overexpressed in chronic myeloid leukemia and interacts with Bcr-Abl

    Experimental Cell Research, Vol. 312, Núm. 6, pp. 938-948

2004

  1. C3G-mediated suppression of oncogene-induced focus formation in fibroblasts involves inhibition of ERK activation, cyclin a expression and alterations of anchorage-independent growth

    Oncogene, Vol. 23, Núm. 28, pp. 4885-4893