INVESTIGACION TRASLACIONAL EN ENFERMEDADES RENALES Y CARDIOVASCULARES
Instituto de Estudios de Ciencias de la Salud de Castilla y León
Soria, EspañaPublications in collaboration with researchers from Instituto de Estudios de Ciencias de la Salud de Castilla y León (31)
2023
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Identification of Pre-Renal and Intrinsic Acute Kidney Injury by Anamnestic and Biochemical Criteria: Distinct Association with Urinary Injury Biomarkers
International journal of molecular sciences, Vol. 24, Núm. 3
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Pathophysiology of Acute Kidney Frailty
Physiology (Bethesda, Md.), Vol. 38, Núm. 6, pp. 0
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Renal sympathetic activity: A key modulator of pressure natriuresis in hypertension
Biochemical Pharmacology, Vol. 208
2022
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Neural Network-Based Calculator for Rat Glomerular Filtration Rate
Biomedicines, Vol. 10, Núm. 3
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RICORS2040: The need for collaborative research in chronic kidney disease
Clinical Kidney Journal, Vol. 15, Núm. 3, pp. 372-387
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The Urinary Level of Injury Biomarkers Is Not Univocally Reflective of the Extent of Toxic Renal Tubular Injury in Rats
International Journal of Molecular Sciences, Vol. 23, Núm. 7
2021
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Cell surface area to volume relationship during apoptosis and apoptotic body formation
Cellular Physiology and Biochemistry, Vol. 55, pp. 161-170
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Regression modeling of the antioxidant-to-nephroprotective relation shows the pivotal role of oxidative stress in cisplatin nephrotoxicity
Antioxidants, Vol. 10, Núm. 9
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The furosemide stress test and computational modeling identify renal damage sites associated with predisposition to acute kidney injury in rats
Translational Research, Vol. 231, pp. 76-91
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The furosemide stress test: Perspectives for acute kidney injury diagnosis
Jornal brasileiro de nefrologia : 'orgao oficial de Sociedades Brasileira e Latino-Americana de Nefrologia
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Urinary Plasminogen Activator Inhibitor-1: A Biomarker of Acute Tubular Injury
American Journal of Nephrology, Vol. 52, Núm. 9, pp. 714-724
2020
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A meta-analysis of preclinical studies using antioxidants for the prevention of cisplatin nephrotoxicity: implications for clinical application
Critical Reviews in Toxicology, Vol. 50, Núm. 9, pp. 780-800
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Combined use of GM2AP and TCP1-eta urinary levels predicts recovery from intrinsic acute kidney injury
Scientific Reports, Vol. 10, Núm. 1
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Impaired Tubular Reabsorption Is the Main Mechanism Explaining Increases in Urinary NGAL Excretion Following Acute Kidney Injury in Rats
Toxicological sciences : an official journal of the Society of Toxicology, Vol. 175, Núm. 1, pp. 75-86
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Metabolic Surgery to Treat Obesity in Diabetic Kidney Disease, Chronic Kidney Disease, and End-Stage Kidney Disease; What Are the Unanswered Questions?
Frontiers in Endocrinology, Vol. 11
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Pathophysiological mechanisms underlying a rat model of triple whammy acute kidney injury
Laboratory Investigation, Vol. 100, Núm. 11, pp. 1455-1464
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Systematic review and meta-analysis of the efficacy of clinically tested protectants of cisplatin nephrotoxicity
European Journal of Clinical Pharmacology, Vol. 76, Núm. 1, pp. 23-33
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Urinary TCP1-eta: A Cortical Damage Marker for the Pathophysiological Diagnosis and Prognosis of Acute Kidney Injury
Toxicological Sciences, Vol. 174, Núm. 1, pp. 3-15
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Urinary transferrin pre-emptively identifies the risk of renal damage posed by subclinical tubular alterations
Biomedicine and Pharmacotherapy, Vol. 121
2018
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N-acetylcysteine transforms necrosis into apoptosis and affords tailored protection from cisplatin cytotoxicity
Toxicology and Applied Pharmacology, Vol. 349, pp. 83-93