Publicaciones en las que colabora con A. Maillo (10)

2007

  1. Alteraciones citogenéticas en meningiomas y su impacto en la evolución de la enfermedad

    Medicina Clinica, Vol. 128, Núm. 6, pp. 226-232

  2. Early recurrences in histologically benign/grade i meningiomas are associated with large tumors and coexistence of monosomy 14 and del(1p36) in the ancestral tumor cell clone

    Neuro-Oncology, Vol. 9, Núm. 4, pp. 438-446

  3. Patient gender is associated with distinct patterns of chromosomal abnormalities and sex chromosome-linked gene-expression profiles in meningiomas

    Oncologist, Vol. 12, Núm. 10, pp. 1225-1236

2006

  1. Microarray-based analysis of spinal versus intracranial meningiomas: Different clinical, biological, and genetic characteristics associated with distinct patterns of gene expression

    Journal of Neuropathology and Experimental Neurology, Vol. 65, Núm. 5, pp. 445-454

  2. The cytogenetic relationship between primary and recurrent meningiomas points to the need for new treatment strategies in cases at high risk of relapse

    Clinical Cancer Research, Vol. 12, Núm. 3 I, pp. 772-780

2005

  1. Characterization of chromosome 14 abnormalities by interphase in situ hybridization and comparative genomic hybridization in 124 meningiomas: Correlation with clinical, histopathologic, and prognostic features

    American Journal of Clinical Pathology, Vol. 123, Núm. 5, pp. 744-751

2004

  1. Intratumoral patterns of clonal evolution in meningiomas as defined by multicolor interphase fluorescence in situ hybridization (FISH): Is there a relationship between histopathologically benign and atypical/anaplastic lesions?

    Journal of Molecular Diagnostics, Vol. 6, Núm. 4, pp. 316-325

2003

  1. New classification scheme for the prognostic stratification of meningioma on the basis of chromosome 14 abnormalities, patient age, and tumor histopathology

    Journal of Clinical Oncology, Vol. 21, Núm. 17, pp. 3285-3295

2002

  1. Incidence of Numerical Chromosome Aberrations in Meningioma Tumors as Revealed by Fluorescence in Situ Hybridization Using 10 Chromosome-Specific Probes

    Clinical Cytometry, Vol. 50, Núm. 3, pp. 153-159

2001

  1. Gains of chromosome 22 by fluorescence in situ hybridization in the context of an hyperdiploid karyotype are associated with aggressive clinical features in meningioma patients

    Cancer, Vol. 92, Núm. 2, pp. 377-385