Publications by the researcher in collaboration with Francisco José López Hernández (44)

2020

  1. Impaired Tubular Reabsorption Is the Main Mechanism Explaining Increases in Urinary NGAL Excretion Following Acute Kidney Injury in Rats

    Toxicological sciences : an official journal of the Society of Toxicology, Vol. 175, Núm. 1, pp. 75-86

  2. Urinary TCP1-eta: A Cortical Damage Marker for the Pathophysiological Diagnosis and Prognosis of Acute Kidney Injury

    Toxicological Sciences, Vol. 174, Núm. 1, pp. 3-15

2019

  1. Cardiotrophin-1 opposes renal fibrosis in mice: Potential prevention of chronic kidney disease

    Acta Physiologica, Vol. 226, Núm. 2

  2. Preventive effect of cardiotrophin-1 administration before DSS-induced ulcerative colitis in mice

    Journal of Clinical Medicine, Vol. 8, Núm. 12

2018

  1. Cardiotrophin-1 Improves Kidney Preservation, Graft Function, and Survival in Transplanted Rats

    Transplantation, Vol. 102, Núm. 10, pp. e404-e412

  2. Cardiotrophin-1 attenuates experimental colitis in mice

    Clinical Science, Vol. 132, Núm. 9, pp. 985-1001

  3. Corrigendum: Association of VAV2 and VAV3 polymorphisms with cardiovascular risk factors

    Scientific reports

  4. N-acetylcysteine transforms necrosis into apoptosis and affords tailored protection from cisplatin cytotoxicity

    Toxicology and Applied Pharmacology, Vol. 349, pp. 83-93

2017

  1. Association of VAV2 and VAV3 polymorphisms with cardiovascular risk factors

    Scientific Reports, Vol. 7

  2. Differential effect of quercetin on cisplatin-induced toxicity in kidney and tumor tissues

    Food and Chemical Toxicology, Vol. 107, pp. 226-236

2016

  1. Cardiotrophin-1 therapy prevents gentamicin-induced nephrotoxicity in rats

    Pharmacological Research, Vol. 107, pp. 137-146

  2. Identification of bone morphogenetic protein 9 (BMP9) as a novel profibrotic factor in vitro

    Cellular Signalling, Vol. 28, Núm. 9, pp. 1252-1261

  3. Mechanisms of triple whammy acute kidney injury

    Pharmacology and Therapeutics, Vol. 167, pp. 132-145

2015

  1. Activation of the ALK-5 pathway is not per se sufficient for the antiproliferative effect of TGF-β1 on renal tubule epithelial cells

    Cellular Physiology and Biochemistry, Vol. 37, Núm. 4, pp. 1231-1239

  2. Acute tubular necrosis: An old term in search for a new meaning within the evolving concept of acute kidney injury

    New Horizons in Translational Medicine, Vol. 2, Núm. 4-5, pp. 110-117

  3. Increased Klk9 urinary excretion is associated to hypertension-induced cardiovascular damage and renal alterations

    Medicine (United States), Vol. 94, Núm. 41

  4. Pathophysiological role of different tubular epithelial cell death modes in acute kidney injury

    Clinical Kidney Journal, Vol. 8, Núm. 5, pp. 548-559

  5. Sub-nephrotoxic cisplatin sensitizes rats to acute renal failure and increases urinary excretion of fumarylacetoacetase

    Toxicology Letters, Vol. 234, Núm. 2, pp. 99-109

  6. Urinary proteomics in renal pathophysiology: Impact of proteinuria

    Proteomics - Clinical Applications, Vol. 9, Núm. 5-6, pp. 636-640

2014

  1. Hypertension and hyperglycemia synergize to cause incipient renal tubular alterations resulting in increased NGAL urinary excretion in rats

    PLoS ONE, Vol. 9, Núm. 8