Víctor Blanco Gozalo-rekin lankidetzan egindako argitalpenak (6)

2020

  1. Combined use of GM2AP and TCP1-eta urinary levels predicts recovery from intrinsic acute kidney injury

    Scientific Reports, Vol. 10, Núm. 1

  2. Impaired Tubular Reabsorption Is the Main Mechanism Explaining Increases in Urinary NGAL Excretion Following Acute Kidney Injury in Rats

    Toxicological sciences : an official journal of the Society of Toxicology, Vol. 175, Núm. 1, pp. 75-86

  3. Pathophysiological mechanisms underlying a rat model of triple whammy acute kidney injury

    Laboratory Investigation, Vol. 100, Núm. 11, pp. 1455-1464

  4. Urinary TCP1-eta: A Cortical Damage Marker for the Pathophysiological Diagnosis and Prognosis of Acute Kidney Injury

    Toxicological Sciences, Vol. 174, Núm. 1, pp. 3-15

2015

  1. Acute tubular necrosis: An old term in search for a new meaning within the evolving concept of acute kidney injury

    New Horizons in Translational Medicine, Vol. 2, Núm. 4-5, pp. 110-117

  2. Sub-nephrotoxic cisplatin sensitizes rats to acute renal failure and increases urinary excretion of fumarylacetoacetase

    Toxicology Letters, Vol. 234, Núm. 2, pp. 99-109