Ana Purificación Velasco Criado-rekin lankidetzan egindako argitalpenak (11)

2018

  1. Endocytosis and Transcytosis of Amyloid-β Peptides by Astrocytes: A Possible Mechanism for Amyloid-β Clearance in Alzheimer's Disease

    Journal of Alzheimer's Disease, Vol. 65, Núm. 4, pp. 1109-1124

2016

  1. Aberrant co-localization of synaptic proteins promoted by Alzheimer's Disease amyloid-β peptides: Protective effect of human serum albumin

    Journal of Alzheimer's Disease, Vol. 55, Núm. 1, pp. 171-182

2009

  1. Albumin endocytosis via megalin in astrocytes is caveola- and Dab-1 dependent and is required for the synthesis of the neurotrophic factor oleic acid

    Journal of Neurochemistry, Vol. 111, Núm. 1, pp. 49-60

  2. La megalina es el receptor para la endocitosis mediada por caveolas de la albúmina y se requiere para la síntesis del factor neurotrófico ácido oleico

    Trauma, Vol. 20, Núm. 3, pp. 130-136

2008

  1. Megalin is a receptor for albumin in astrocytes and is required for the synthesis of the neurotrophic factor oleic acid

    Journal of Neurochemistry, Vol. 106, Núm. 3, pp. 1149-1159

2004

  1. The neurotrophic effect of oleic acid includes dendritic differentiation and the expression of the neuronal basic helix-loop-helix transcription factor NeuroD2

    Journal of Neurochemistry, Vol. 88, Núm. 5, pp. 1041-1051

2003

  1. Role of oleic acid as a neurotrophic factor is supported in vivo by the expression of GAP-43 subsequent to the activation of SREBP-1 and the up-regulation of stearoyl-CoA desaturase during postnatal development of the brain

    Brain Research, Vol. 977, Núm. 1, pp. 103-111

2002

  1. Transcytosis of albumin in astrocytes activates the sterol regulatory element-binding protein-1, which promotes the synthesis of the neurotrophic factor oleic acid

    Journal of Biological Chemistry, Vol. 277, Núm. 6, pp. 4240-4246

2001

  1. Neuronal differentiation is triggered by oleic acid synthesized and released by astrocytes

    Journal of Neurochemistry, Vol. 79, Núm. 3, pp. 606-616

  2. The enhancement of glucose uptake caused by the collapse of gap junction communication is due to an increase in astrocyte proliferation

    Journal of Neurochemistry, Vol. 78, Núm. 4, pp. 890-898

2000

  1. ATP-Sensitive potassium channel regulates astrocytic gap junction permeability by a Ca2+-independent mechanism

    Journal of Neurochemistry, Vol. 74, Núm. 3, pp. 1249-1256