Publicaciones en colaboración con investigadores/as de Instituto de Investigación Biomédica de Salamanca (18)

2024

  1. Combined HRAS and NRAS ablation induces a RASopathy phenotype in mice

    Cell Communication and Signaling, Vol. 22, Núm. 1

  2. Platelet C3G: a key player in vesicle exocytosis, spreading and clot retraction

    Cellular and Molecular Life Sciences, Vol. 81, Núm. 1

2023

  1. Additional file 3 of Crk proteins activate the Rap1 guanine nucleotide exchange factor C3G by segregated adaptor-dependent and -independent mechanisms

    Additional file 3 of Crk proteins activate the Rap1 guanine nucleotide exchange factor C3G by segregated adaptor-dependent and -independent mechanisms

  2. Additional file 3 of Crk proteins activate the Rap1 guanine nucleotide exchange factor C3G by segregated adaptor-dependent and -independent mechanisms

    Additional file 3 of Crk proteins activate the Rap1 guanine nucleotide exchange factor C3G by segregated adaptor-dependent and -independent mechanisms

  3. Crk proteins activate the Rap1 guanine nucleotide exchange factor C3G by segregated adaptor-dependent and -independent mechanisms

    Cell Communication and Signaling, Vol. 21, Núm. 1

2022

  1. C3G down-regulation enhances pro-migratory and stemness properties of oval cells by promoting an epithelial-mesenchymal-like process

    International journal of biological sciences, Vol. 18, Núm. 15, pp. 5873-5884

  2. New functions of C3G in platelet biology: Contribution to ischemia-induced angiogenesis, tumor metastasis and TPO clearance

    Frontiers in Cell and Developmental Biology, Vol. 10

2021

  1. C3G downregulation induces the acquisition of a mesenchymal phenotype that enhances aggressiveness of glioblastoma cells

    Cell Death and Disease, Vol. 12, Núm. 4

  2. C3G protein, a new player in glioblastoma

    International Journal of Molecular Sciences, Vol. 22, Núm. 18

  3. Contribution of c3g and other gefs to liver cancer development and progression

    Hepatoma Research, Vol. 7

2020

  1. C3G contributes to platelet activation and aggregation by regulating major signaling pathways

    Signal Transduction and Targeted Therapy, Vol. 5, Núm. 1

  2. C3G is upregulated in hepatocarcinoma, contributing to tumor growth and progression and to HGF/MET pathway activation

    Cancers, Vol. 12, Núm. 8, pp. 1-22

2018

  1. C3G, through its GEF activity, induces megakaryocytic differentiation and proplatelet formation

    Cell Communication and Signaling, Vol. 16, Núm. 1

2017

  1. C3G promotes a selective release of angiogenic factors from activated mouse platelets to regulate angiogenesis and tumor metastasis

    Oncotarget, Vol. 8, Núm. 67, pp. 110994-111011

  2. The CRISPR/Cas9 system efficiently reverts the tumorigenic ability of BCR/ABL in vitro and in a xenograft model of chronic myeloid leukemia

    Oncotarget, Vol. 8, Núm. 16, pp. 26027-26040

2015

  1. Incidence and risk factors for life-threatening bleeding after allogeneic stem cell transplant

    British Journal of Haematology, Vol. 169, Núm. 5, pp. 719-725

  2. p38 MAPK down-regulates fibulin 3 expression through methylation of gene regulatory sequences: Role in migration and invasion

    Journal of Biological Chemistry, Vol. 290, Núm. 7, pp. 4383-4397

2012

  1. C3G transgenic mouse models with specific expression in platelets reveal a new role for C3G in platelet clotting through its GEF activity

    Biochimica et Biophysica Acta - Molecular Cell Research, Vol. 1823, Núm. 8, pp. 1366-1377