María Begoña
García Cenador
Profesora Titular de Universidad
![Foto de María Begoña](/img/nophoto.png)
![Foto de Instituto de Biología Molecular y Celular del Cáncer de Salamanca / Centro de Investigación del Cáncer](/img/noimage_org.png)
Instituto de Biología Molecular y Celular del Cáncer de Salamanca / Centro de Investigación del Cáncer
Salamanca, EspañaPublicaciones en colaboración con investigadores/as de Instituto de Biología Molecular y Celular del Cáncer de Salamanca / Centro de Investigación del Cáncer (13)
2022
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Transient Inhibition of the JAK/STAT Pathway Prevents B-ALL Development in Genetically Predisposed Mice
Cancer research, Vol. 82, Núm. 6, pp. 1098-1109
2020
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An intact gut microbiome protects genetically predisposed mice against leukemia
Blood, Vol. 136, Núm. 18, pp. 2003-2017
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Inhibition of inflammatory signaling in Pax5 mutant cells mitigates B-cell leukemogenesis
Scientific Reports, Vol. 10, Núm. 1
2019
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Dnmt1 links BCR-ABLp210 to epigenetic tumor stem cell priming in myeloid leukemia
Leukemia, Vol. 33, Núm. 1, pp. 249-278
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Infectious stimuli promote malignant B-cell acute lymphoblastic leukemia in the absence of AID
Nature Communications, Vol. 10, Núm. 1
2018
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Lmo2 expression defines tumor cell identity during T-cell leukemogenesis
EMBO Journal, Vol. 37, Núm. 14
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Loss of Pax5 exploits sca1-BCR-ABLp190 susceptibility to confer the metabolic shift essential for pB-ALL
Cancer Research, Vol. 78, Núm. 10, pp. 2669-2679
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Supplementary data for the biological age linked to oxidative stress modifies breast cancer aggressiveness
Data in Brief
2017
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Infection exposure promotes ETV6-RUNX1 precursor B-cell leukemia via impaired H3K4 demethylases
Cancer Research, Vol. 77, Núm. 16, pp. 4365-4377
2015
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Infection exposure is a causal factor in B-cell precursor acute lymphoblastic leukemia as a result of Pax5-inherited susceptibility
Cancer Discovery, Vol. 5, Núm. 12, pp. 1328-1343
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Unraveling heterogeneous susceptibility and the evolution of breast cancer using a systems biology approach
Genome Biology, Vol. 16, Núm. 1
2012
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A novel molecular mechanism involved in multiple myeloma development revealed by targeting MafB to haematopoietic progenitors
EMBO Journal, Vol. 31, Núm. 18, pp. 3704-3717
2010
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Cancer as a reprogramming-like disease: Implications in tumor development and treatment
Seminars in Cancer Biology, Vol. 20, Núm. 2, pp. 93-97