JOSÉ MIGUEL LÓPEZ NOVOA-rekin lankidetzan egindako argitalpenak (29)

2020

  1. Impaired Tubular Reabsorption Is the Main Mechanism Explaining Increases in Urinary NGAL Excretion Following Acute Kidney Injury in Rats

    Toxicological sciences : an official journal of the Society of Toxicology, Vol. 175, Núm. 1, pp. 75-86

2015

  1. Acute tubular necrosis: An old term in search for a new meaning within the evolving concept of acute kidney injury

    New Horizons in Translational Medicine, Vol. 2, Núm. 4-5, pp. 110-117

  2. Plasma cardiotrophin-1 as a marker of hypertension and diabetes-induced target organ damage and cardiovascular risk

    Medicine (United States), Vol. 94, Núm. 30

2014

  1. L-endoglin overexpression increases renal fibrosis after unilateral ureteral obstruction

    PLoS ONE, Vol. 9, Núm. 10

2011

  1. net Etiopathology of chronic tubular, glomerular and renovascular nephropathies: Clinical implications

    Journal of Translational Medicine, Vol. 9

2010

  1. Analysis of K-Ras nuclear expression in fibroblasts and mesangial cells

    PLoS ONE, Vol. 5, Núm. 1

  2. Common pathophysiological mechanisms of chronic kidney disease: Therapeutic perspectives

    Pharmacology and Therapeutics, Vol. 128, Núm. 1, pp. 61-81

  3. Deletion of H-Ras decreases renal fibrosis and myofibroblast activation following ureteral obstruction in mice

    Kidney International, Vol. 77, Núm. 6, pp. 509-518

  4. Increased plasma soluble endoglin levels as an indicator of cardiovascular alterations in hypertensive and diabetic patients

    BMC Medicine, Vol. 8

  5. Mechanisms involved in the genesis of diabetic nephropathy

    Current Diabetes Reviews, Vol. 6, Núm. 2, pp. 68-87

2009

  1. Effect of different antihypertensive treatments on Ras, MAPK and Akt activation in hypertension and diabetes

    Clinical Science, Vol. 116, Núm. 2, pp. 165-173

2008

  1. Involvement of small Ras GTPases and their effectors in chronic renal disease

    Cellular and Molecular Life Sciences, Vol. 65, Núm. 3, pp. 477-492

2007

  1. Effect of adenosine in extracellular matrix synthesis in human and rat mesangial cells

    Molecular and Cellular Biochemistry, Vol. 305, Núm. 1-2, pp. 163-169

  2. Glomerular nephrotoxicity of aminoglycosides

    Toxicology and Applied Pharmacology, Vol. 223, Núm. 1, pp. 86-98

2006

  1. Involvement of H- and N-Ras isoforms in transforming growth factor-β1-induced proliferation and in collagen and fibronectin synthesis

    Experimental Cell Research, Vol. 312, Núm. 11, pp. 2093-2106

2005

  1. Gentamicin induces Jun-AP1 expression and JNK activation in renal glomeruli and cultured mesangial cells

    Life Sciences, Vol. 77, Núm. 18, pp. 2285-2298

2004

  1. Erratum: Gentamicin treatment induces simultaneous mesangial proliferation and apoptosis in rats (Kidney International (2004) 65 (2161-2171))

    Kidney International

  2. Gentamicin treatment induces simultaneous mesangial proliferation and apoptosis in rats

    Kidney International, Vol. 65, Núm. 6, pp. 2161-2171

2003

  1. Effect of ham protein substitution on oxidative stress in older adults

    Journal of Nutrition, Health and Aging, Vol. 7, Núm. 2, pp. 84-89

2002

  1. Cyclosporin effect on rat aorta α1-adrenoceptors and their transduction mechanisms

    Journal of Cardiovascular Pharmacology, Vol. 40, Núm. 2, pp. 181-188