Víctor Blanco Gozalo-rekin lankidetzan egindako argitalpenak (7)

2020

  1. Combined use of GM2AP and TCP1-eta urinary levels predicts recovery from intrinsic acute kidney injury

    Scientific Reports, Vol. 10, Núm. 1

  2. Impaired Tubular Reabsorption Is the Main Mechanism Explaining Increases in Urinary NGAL Excretion Following Acute Kidney Injury in Rats

    Toxicological sciences : an official journal of the Society of Toxicology, Vol. 175, Núm. 1, pp. 75-86

  3. Pathophysiological mechanisms underlying a rat model of triple whammy acute kidney injury

    Laboratory Investigation, Vol. 100, Núm. 11, pp. 1455-1464

  4. Urinary TCP1-eta: A Cortical Damage Marker for the Pathophysiological Diagnosis and Prognosis of Acute Kidney Injury

    Toxicological Sciences, Vol. 174, Núm. 1, pp. 3-15

2015

  1. Acute tubular necrosis: An old term in search for a new meaning within the evolving concept of acute kidney injury

    New Horizons in Translational Medicine, Vol. 2, Núm. 4-5, pp. 110-117

  2. Increased Klk9 urinary excretion is associated to hypertension-induced cardiovascular damage and renal alterations

    Medicine (United States), Vol. 94, Núm. 41

2014

  1. Hypertension and hyperglycemia synergize to cause incipient renal tubular alterations resulting in increased NGAL urinary excretion in rats

    PLoS ONE, Vol. 9, Núm. 8