Publikationen (91) Publikationen von José Carlos Martínez Salgado

2023

  1. Identification of Pre-Renal and Intrinsic Acute Kidney Injury by Anamnestic and Biochemical Criteria: Distinct Association with Urinary Injury Biomarkers

    International journal of molecular sciences, Vol. 24, Núm. 3

  2. Phosphorus May Induce Phenotypic Transdifferentiation of Vascular Smooth Muscle Cells through the Reduction of microRNA-145

    Nutrients, Vol. 15, Núm. 13

  3. Renal sympathetic activity: A key modulator of pressure natriuresis in hypertension

    Biochemical Pharmacology, Vol. 208

2022

  1. Endothelial Activin Receptor-Like Kinase 1 (ALK1) Regulates Myofibroblast Emergence and Peritubular Capillary Stability in the Early Stages of Kidney Fibrosis

    Frontiers in Pharmacology, Vol. 13

  2. Neural Network-Based Calculator for Rat Glomerular Filtration Rate

    Biomedicines, Vol. 10, Núm. 3

  3. Retinal blood vessel calibre and vascular ageing in a general Spanish population: A EVA study

    European Journal of Clinical Investigation, Vol. 52, Núm. 2

  4. Urinary KIM-1 Correlates with the Subclinical Sequelae of Tubular Damage Persisting after the Apparent Functional Recovery from Intrinsic Acute Kidney Injury

    Biomedicines, Vol. 10, Núm. 5

2021

  1. Albuminuria pre-emptively identifies cardiac patients at risk of contrast-induced nephropathy

    Journal of Clinical Medicine, Vol. 10, Núm. 21

  2. Biomarkers of persistent renal vulnerability after acute kidney injury recovery

    Scientific Reports, Vol. 11, Núm. 1

  3. Dissecting the Involvement of Ras GTPases in Kidney Fibrosis

    Genes, Vol. 12, Núm. 6

  4. Haemodynamic frailty – A risk factor for acute kidney injury in the elderly

    Ageing Research Reviews, Vol. 70

  5. Sos1 Modulates Extracellular Matrix Synthesis, Proliferation, and Migration in Fibroblasts

    Frontiers in Physiology, Vol. 12

  6. The furosemide stress test and computational modeling identify renal damage sites associated with predisposition to acute kidney injury in rats

    Translational Research, Vol. 231, pp. 76-91

2020

  1. A meta-analysis of preclinical studies using antioxidants for the prevention of cisplatin nephrotoxicity: implications for clinical application

    Critical Reviews in Toxicology, Vol. 50, Núm. 9, pp. 780-800

  2. Association of Alk1 and Endoglin Polymorphisms with Cardiovascular Damage

    Scientific Reports, Vol. 10, Núm. 1

  3. Combined use of GM2AP and TCP1-eta urinary levels predicts recovery from intrinsic acute kidney injury

    Scientific Reports, Vol. 10, Núm. 1

  4. Impaired Tubular Reabsorption Is the Main Mechanism Explaining Increases in Urinary NGAL Excretion Following Acute Kidney Injury in Rats

    Toxicological sciences : an official journal of the Society of Toxicology, Vol. 175, Núm. 1, pp. 75-86

  5. Pathophysiological mechanisms underlying a rat model of triple whammy acute kidney injury

    Laboratory Investigation, Vol. 100, Núm. 11, pp. 1455-1464

  6. Reference values of arterial stiffness parameters and their association with cardiovascular risk factors in the Spanish population. The EVA Study

    Revista Espanola de Cardiologia, Vol. 73, Núm. 1, pp. 43-52

  7. Systematic review and meta-analysis of the efficacy of clinically tested protectants of cisplatin nephrotoxicity

    European Journal of Clinical Pharmacology, Vol. 76, Núm. 1, pp. 23-33