BIOLOGÍA CELULAR Y PATOLOGÍA
Departamento
JESÚS FERNANDO
SAN MIGUEL IZQUIERDO
Investigador en el periodo 1986-2013
Publicaciones en las que colabora con JESÚS FERNANDO SAN MIGUEL IZQUIERDO (13)
2016
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Targeting of PI3K/AKT/mTOR pathway to inhibit T cell activation and prevent graft-versus-host disease development
Journal of Hematology and Oncology, Vol. 9, Núm. 1
2015
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Phenotypic identification of subclones in multiple myeloma with different chemoresistant, cytogenetic and clonogenic potential
Leukemia, Vol. 29, Núm. 5, pp. 1186-1194
2013
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Detailed characterization of multiple myeloma circulating tumor cells shows unique phenotypic, cytogenetic, functional, and circadian distribution profile
Blood, Vol. 122, Núm. 22, pp. 3591-3598
2012
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Dasatinib as a bone-modifying agent: Anabolic and anti-resorptive effects
PLoS ONE, Vol. 7, Núm. 4
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SNP-based mapping arrays reveal high genomic complexity in monoclonal gammopathies, from MGUS to myeloma status
Leukemia, Vol. 26, Núm. 12, pp. 2521-2529
2010
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Both CD133+ cells and monocytes provide significant improvement for hindlimb ischemia, although they do not transdifferentiate into endothelial cells
Cell Transplantation, Vol. 19, Núm. 1, pp. 103-112
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Isolation and characterization of mesenchymal stromal cells from human degenerated nucleus pulposus: Comparison with bone marrow mesenchymal stromal cells from the same subjects
Spine, Vol. 35, Núm. 26, pp. 2259-2265
2006
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Heterogeneity of neoplastic cells in B-cell chronic lymphoproliferative disorders: Biclonality versus intraclonal evolution of a single tumor cell clone
Haematologica, Vol. 91, Núm. 3, pp. 331-339
2005
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Efficacy of rituximab in an aggressive form of multicentric Castleman disease associated with immune phenomena
American Journal of Hematology, Vol. 78, Núm. 4, pp. 302-305
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Genetic heterogeneity of BCR/ABL+ adult B-cell precursor acute lymphoblastic leukemia: Impact on the clinical, biological and immunophenotypical disease characteristics
Leukemia, Vol. 19, Núm. 5, pp. 713-720
2003
2001
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Adult precursor B-ALL with BCR/ABL gene rearrangements displays a unique immunophenotype based on the pattern of CD10, CD34, CD13 and CD38 expression
Leukemia, Vol. 15, Núm. 3, pp. 406-414