Piero Crespo-rekin lankidetzan egindako argitalpenak (22)

2022

  1. Immune Checkpoint Inhibitors and RAS–ERK Pathway-Targeted Drugs as Combined Therapy for the Treatment of Melanoma

    Biomolecules, Vol. 12, Núm. 11

  2. The Rho guanosine nucleotide exchange factors Vav2 and Vav3 modulate epidermal stem cell function

    Oncogene, Vol. 41, Núm. 24, pp. 3341-3354

2020

  1. RAC1 induces nuclear alterations through the LINC complex to enhance melanoma invasiveness

    Molecular biology of the cell, Vol. 31, Núm. 25, pp. 2768-2778

2019

  1. Corrigendum to: Vav mediates Ras stimulation by direct activation of the GDP/GTP exchange factor Ras GRP1 (The EMBO Journal, (2003), 22, 13, (3326-3336), 10.1093/emboj/cdg316)

    EMBO Journal

2018

  1. Protein–Protein Interactions: Emerging Oncotargets in the RAS-ERK Pathway

    Trends in Cancer, Vol. 4, Núm. 9, pp. 616-633

  2. RAS GTPase-dependent pathways in developmental diseases: old guys, new lads, and current challenges

    Current Opinion in Cell Biology, Vol. 55, pp. 42-51

  3. RAS at the Golgi antagonizes malignant transformation through PTPRκ-mediated inhibition of ERK activation

    Nature Communications, Vol. 9, Núm. 1

  4. The RAS-ERK pathway: A route for couples

    Science Signaling, Vol. 11, Núm. 554

2016

  1. PGA1-induced apoptosis involves specific activation of H-Ras and N-Ras in cellular endomembranes

    Cell Death and Disease, Vol. 7, Núm. 7

2013

  1. The small GTPase N-Ras regulates extracellular matrix synthesis, proliferation and migration in fibroblasts

    Biochimica et Biophysica Acta - Molecular Cell Research, Vol. 1833, Núm. 12, pp. 2734-2744

2007

  1. Transcriptomal profiling of site-specific Ras signals

    Cellular Signalling, Vol. 19, Núm. 11, pp. 2264-2276

2006

  1. Distinct utilization of effectors and biological outcomes resulting from site-specific ras activation: Ras functions in lipid rafts and golgi complex are dispensable for proliferation and transformation

    Molecular and Cellular Biology, Vol. 26, Núm. 1, pp. 100-116

2003

  1. Vav mediates Ras stimulation by direct activation of the GDP/GTP exchange factor Ras GRP1

    EMBO Journal, Vol. 22, Núm. 13, pp. 3326-3336

2002

  1. Erk5 participates in neuregulin signal transduction and is constitutively active in breast cancer cells overexpressing ErbB2

    Molecular and Cellular Biology, Vol. 22, Núm. 1, pp. 270-285

  2. Walking pathways at Heidelberg. Conference on oncogenes and growth control

    EMBO Reports

1999

  1. Identification and chromosomal location of two human genes encoding enzymes potentially involved in proteolytic maturation of farnesylated proteins

    Genomics, Vol. 58, Núm. 3, pp. 270-280

  2. Signal transduction elements of TC21, an oncogenic member of the R-Ras subfamily of GTP-binding proteins

    Oncogene, Vol. 18, Núm. 43, pp. 5860-5869

1998

  1. El oncogén "vav"

    Investigación y ciencia, Núm. 262, pp. 62-71

  2. Transforming G protein-coupled receptors block insulin and ras-induced adipocytic differentiation in 3T3-L1 cells: Evidence for a PKC and MAP kinase independent pathway

    Biochemical and Biophysical Research Communications, Vol. 245, Núm. 2, pp. 554-561

1997

  1. Cbl-b, a member of the Sli-1/c-Cbl protein family, inhibits Vav-mediated c-Jun N-terminal kinase activation

    Oncogene, Vol. 15, Núm. 21, pp. 2511-2520