Publicacións nas que colabora con Francisco José López Hernández (39)

2024

  1. Effect of uric acid reduction on chronic kidney disease. Systematic review and meta-analysis

    Frontiers in Pharmacology, Vol. 15

2023

  1. Identification of Pre-Renal and Intrinsic Acute Kidney Injury by Anamnestic and Biochemical Criteria: Distinct Association with Urinary Injury Biomarkers

    International journal of molecular sciences, Vol. 24, Núm. 3

2022

  1. Diagnosis of Cardiac Surgery-Associated Acute Kidney Injury: State of the Art and Perspectives

    Journal of Clinical Medicine, Vol. 11, Núm. 15

  2. Neural Network-Based Calculator for Rat Glomerular Filtration Rate

    Biomedicines, Vol. 10, Núm. 3

  3. Protective Effect of Quercetin 3-O-Glucuronide against Cisplatin Cytotoxicity in Renal Tubular Cells

    Molecules, Vol. 27, Núm. 4

2021

  1. A micellar formulation of quercetin prevents cisplatin nephrotoxicity

    International Journal of Molecular Sciences, Vol. 22, Núm. 2, pp. 1-14

  2. Albuminuria pre-emptively identifies cardiac patients at risk of contrast-induced nephropathy

    Journal of Clinical Medicine, Vol. 10, Núm. 21

  3. Are antioxidants useful in preventing the progression of chronic kidney disease?

    Antioxidants, Vol. 10, Núm. 11

  4. Regression modeling of the antioxidant-to-nephroprotective relation shows the pivotal role of oxidative stress in cisplatin nephrotoxicity

    Antioxidants, Vol. 10, Núm. 9

  5. The furosemide stress test and computational modeling identify renal damage sites associated with predisposition to acute kidney injury in rats

    Translational Research, Vol. 231, pp. 76-91

  6. Urinary Plasminogen Activator Inhibitor-1: A Biomarker of Acute Tubular Injury

    American Journal of Nephrology, Vol. 52, Núm. 9, pp. 714-724

2020

  1. A meta-analysis of preclinical studies using antioxidants for the prevention of cisplatin nephrotoxicity: implications for clinical application

    Critical Reviews in Toxicology, Vol. 50, Núm. 9, pp. 780-800

  2. Combined use of GM2AP and TCP1-eta urinary levels predicts recovery from intrinsic acute kidney injury

    Scientific Reports, Vol. 10, Núm. 1

  3. Impaired Tubular Reabsorption Is the Main Mechanism Explaining Increases in Urinary NGAL Excretion Following Acute Kidney Injury in Rats

    Toxicological sciences : an official journal of the Society of Toxicology, Vol. 175, Núm. 1, pp. 75-86

  4. Pathophysiological mechanisms underlying a rat model of triple whammy acute kidney injury

    Laboratory Investigation, Vol. 100, Núm. 11, pp. 1455-1464

  5. Systematic review and meta-analysis of the efficacy of clinically tested protectants of cisplatin nephrotoxicity

    European Journal of Clinical Pharmacology, Vol. 76, Núm. 1, pp. 23-33

  6. Urinary TCP1-eta: A Cortical Damage Marker for the Pathophysiological Diagnosis and Prognosis of Acute Kidney Injury

    Toxicological Sciences, Vol. 174, Núm. 1, pp. 3-15

  7. Urinary transferrin pre-emptively identifies the risk of renal damage posed by subclinical tubular alterations

    Biomedicine and Pharmacotherapy, Vol. 121

2019

  1. Cardiotrophin-1 opposes renal fibrosis in mice: Potential prevention of chronic kidney disease

    Acta Physiologica, Vol. 226, Núm. 2

  2. Quercetin, a promising clinical candidate for the prevention of contrast-induced nephropathy

    International Journal of Molecular Sciences, Vol. 20, Núm. 19