Publicaciones en colaboración con investigadores/as de Instituto de Estudios de Ciencias de la Salud de Castilla y León (19)

2023

  1. Identification of Pre-Renal and Intrinsic Acute Kidney Injury by Anamnestic and Biochemical Criteria: Distinct Association with Urinary Injury Biomarkers

    International journal of molecular sciences, Vol. 24, Núm. 3

2022

  1. Neural Network-Based Calculator for Rat Glomerular Filtration Rate

    Biomedicines, Vol. 10, Núm. 3

2021

  1. Regression modeling of the antioxidant-to-nephroprotective relation shows the pivotal role of oxidative stress in cisplatin nephrotoxicity

    Antioxidants, Vol. 10, Núm. 9

  2. The furosemide stress test and computational modeling identify renal damage sites associated with predisposition to acute kidney injury in rats

    Translational Research, Vol. 231, pp. 76-91

  3. Urinary Plasminogen Activator Inhibitor-1: A Biomarker of Acute Tubular Injury

    American Journal of Nephrology, Vol. 52, Núm. 9, pp. 714-724

2020

  1. A meta-analysis of preclinical studies using antioxidants for the prevention of cisplatin nephrotoxicity: implications for clinical application

    Critical Reviews in Toxicology, Vol. 50, Núm. 9, pp. 780-800

  2. Combined use of GM2AP and TCP1-eta urinary levels predicts recovery from intrinsic acute kidney injury

    Scientific Reports, Vol. 10, Núm. 1

  3. Impaired Tubular Reabsorption Is the Main Mechanism Explaining Increases in Urinary NGAL Excretion Following Acute Kidney Injury in Rats

    Toxicological sciences : an official journal of the Society of Toxicology, Vol. 175, Núm. 1, pp. 75-86

  4. Pathophysiological mechanisms underlying a rat model of triple whammy acute kidney injury

    Laboratory Investigation, Vol. 100, Núm. 11, pp. 1455-1464

  5. Systematic review and meta-analysis of the efficacy of clinically tested protectants of cisplatin nephrotoxicity

    European Journal of Clinical Pharmacology, Vol. 76, Núm. 1, pp. 23-33

  6. Urinary TCP1-eta: A Cortical Damage Marker for the Pathophysiological Diagnosis and Prognosis of Acute Kidney Injury

    Toxicological Sciences, Vol. 174, Núm. 1, pp. 3-15

  7. Urinary transferrin pre-emptively identifies the risk of renal damage posed by subclinical tubular alterations

    Biomedicine and Pharmacotherapy, Vol. 121

2018

  1. N-acetylcysteine transforms necrosis into apoptosis and affords tailored protection from cisplatin cytotoxicity

    Toxicology and Applied Pharmacology, Vol. 349, pp. 83-93

2017

  1. A systematic meta-analysis on the efficacy of pre-clinically tested nephroprotectants at preventing aminoglycoside nephrotoxicity

    Toxicology, Vol. 377, pp. 14-24

  2. Differential effect of quercetin on cisplatin-induced toxicity in kidney and tumor tissues

    Food and Chemical Toxicology, Vol. 107, pp. 226-236

2015

  1. Sub-nephrotoxic cisplatin sensitizes rats to acute renal failure and increases urinary excretion of fumarylacetoacetase

    Toxicology Letters, Vol. 234, Núm. 2, pp. 99-109

2013

  1. Cardiotrophin-1 administration prevents the renal toxicity of iodinated contrast media in rats

    Toxicological Sciences, Vol. 132, Núm. 2, pp. 493-501

  2. Increased urinary excretion of albumin, hemopexin, transferrin and VDBP correlates with chronic sensitization to gentamicin nephrotoxicity in rats

    Toxicology, Vol. 304, pp. 83-91

2011

  1. An integrative view of the pathophysiological events leading to cisplatin nephrotoxicity

    Critical Reviews in Toxicology, Vol. 41, Núm. 10, pp. 803-821