Publicaciones en las que colabora con Juan Pedro Bolaños Hernández (24)

2023

  1. Fatty acid oxidation organizes mitochondrial supercomplexes to sustain astrocytic ROS and cognition

    Nature Metabolism, Vol. 5, Núm. 8, pp. 1290-1302

2022

  1. Aberrant upregulation of the glycolytic enzyme PFKFB3 in CLN7 neuronal ceroid lipofuscinosis

    Nature Communications, Vol. 13, Núm. 1

2021

  1. Opa1 relies on cristae preservation and ATP synthase to curtail reactive oxygen species accumulation in mitochondria

    Redox Biology, Vol. 41

2019

  1. Astrocytic mitochondrial ROS modulate brain metabolism and mouse behaviour

    Nature Metabolism

2018

  1. NF-κB Activity Initiates Human ESC-Derived Neural Progenitor Cell Differentiation by Inducing a Metabolic Maturation Program

    Stem Cell Reports, Vol. 10, Núm. 6, pp. 1766-1781

2017

  1. Erratum to: Inflammation, glucose, and vascular cell damage: The role of the pentose phosphate pathway. [Cardiovasc Diabetol. 15, (2016), (82)] DOI: 10.1186/s12933-016-0397-2

    Cardiovascular Diabetology

2016

  1. Inflammation, glucose, and vascular cell damage: The role of the pentose phosphate pathway

    Cardiovascular Diabetology, Vol. 15, Núm. 1

  2. NRF2 Orchestrates the Metabolic Shift during Induced Pluripotent Stem Cell Reprogramming

    Cell Reports, Vol. 14, Núm. 8, pp. 1883-1891

  3. α-Ketoglutarate dehydrogenase complex moonlighting: ROS signalling added to the list: An Editorial highlight for ‘Reductions in the mitochondrial enzyme α-ketoglutarate dehydrogenase complex in neurodegenerative disease - beneficial or detrimental?’

    Journal of Neurochemistry

2015

  1. DJ1 represses glycolysis and cell proliferation by transcriptionally up-regulating Pink1

    Biochemical Journal, Vol. 467, Núm. 2, pp. 303-310

2014

  1. Abstract 560: Inflammation Converts Glucose Into A Deleterious Agent In Human Aortic Smooth Muscle Cells

    Hypertension, Vol. 64, Núm. suppl_1

  2. Pink1 deficiency sustains cell proliferation by reprogramming glucose metabolism through hif1

    Nature Communications, Vol. 5

  3. The oxidized form of vitamin C, dehydroascorbic acid, regulates neuronal energy metabolism

    Journal of Neurochemistry, Vol. 129, Núm. 4, pp. 663-671

2013

  1. Underestimation of the pentose-phosphate pathway in intact primary neurons as revealed by metabolic flux analysis

    Journal of Cerebral Blood Flow and Metabolism, Vol. 33, Núm. 12, pp. 1843-1845

2009

  1. The bioenergetic and antioxidant status of neurons is controlled by continuous degradation of a key glycolytic enzyme by APC/C-Cdh1

    Nature Cell Biology, Vol. 11, Núm. 6, pp. 747-752

2007

  1. Reduced tumor growth and angiogenesis in endoglin-haploinsufficient mice

    Tumor Biology, Vol. 28, Núm. 1, pp. 1-8

2005

  1. Inhibition of mitochondrial respiration by nitric oxide: Its role in glucose metabolism and neuroprotection

    Journal of Neuroscience Research

  2. Knockdown of glutamate-cysteine by ligase by small hairpin RNA reveals that both catalytic and modulatory subunits are essential for the survival of primary neurons

    Journal of Biological Chemistry, Vol. 280, Núm. 47, pp. 38992-39001

2004

  1. Regulation of glucose metabolism by nitrosative stress in neural cells

    Molecular Aspects of Medicine, Vol. 25, Núm. 1-2, pp. 61-73

2002

  1. Involvement of reactive oxygen species on gentamicin-induced mesangial cell activation

    Kidney International, Vol. 62, Núm. 5, pp. 1682-1692