Publicaciones en las que colabora con JOSÉ MIGUEL LÓPEZ NOVOA (29)

2020

  1. Impaired Tubular Reabsorption Is the Main Mechanism Explaining Increases in Urinary NGAL Excretion Following Acute Kidney Injury in Rats

    Toxicological sciences : an official journal of the Society of Toxicology, Vol. 175, Núm. 1, pp. 75-86

2015

  1. Acute tubular necrosis: An old term in search for a new meaning within the evolving concept of acute kidney injury

    New Horizons in Translational Medicine, Vol. 2, Núm. 4-5, pp. 110-117

  2. Plasma cardiotrophin-1 as a marker of hypertension and diabetes-induced target organ damage and cardiovascular risk

    Medicine (United States), Vol. 94, Núm. 30

2014

  1. L-endoglin overexpression increases renal fibrosis after unilateral ureteral obstruction

    PLoS ONE, Vol. 9, Núm. 10

2011

  1. net Etiopathology of chronic tubular, glomerular and renovascular nephropathies: Clinical implications

    Journal of Translational Medicine, Vol. 9

2010

  1. Analysis of K-Ras nuclear expression in fibroblasts and mesangial cells

    PLoS ONE, Vol. 5, Núm. 1

  2. Common pathophysiological mechanisms of chronic kidney disease: Therapeutic perspectives

    Pharmacology and Therapeutics, Vol. 128, Núm. 1, pp. 61-81

  3. Deletion of H-Ras decreases renal fibrosis and myofibroblast activation following ureteral obstruction in mice

    Kidney International, Vol. 77, Núm. 6, pp. 509-518

  4. Increased plasma soluble endoglin levels as an indicator of cardiovascular alterations in hypertensive and diabetic patients

    BMC Medicine, Vol. 8

  5. Mechanisms involved in the genesis of diabetic nephropathy

    Current Diabetes Reviews, Vol. 6, Núm. 2, pp. 68-87

2009

  1. Effect of different antihypertensive treatments on Ras, MAPK and Akt activation in hypertension and diabetes

    Clinical Science, Vol. 116, Núm. 2, pp. 165-173

2008

  1. Involvement of small Ras GTPases and their effectors in chronic renal disease

    Cellular and Molecular Life Sciences, Vol. 65, Núm. 3, pp. 477-492

2007

  1. Effect of adenosine in extracellular matrix synthesis in human and rat mesangial cells

    Molecular and Cellular Biochemistry, Vol. 305, Núm. 1-2, pp. 163-169

  2. Glomerular nephrotoxicity of aminoglycosides

    Toxicology and Applied Pharmacology, Vol. 223, Núm. 1, pp. 86-98

2006

  1. Involvement of H- and N-Ras isoforms in transforming growth factor-β1-induced proliferation and in collagen and fibronectin synthesis

    Experimental Cell Research, Vol. 312, Núm. 11, pp. 2093-2106

2005

  1. Gentamicin induces Jun-AP1 expression and JNK activation in renal glomeruli and cultured mesangial cells

    Life Sciences, Vol. 77, Núm. 18, pp. 2285-2298

2004

  1. Erratum: Gentamicin treatment induces simultaneous mesangial proliferation and apoptosis in rats (Kidney International (2004) 65 (2161-2171))

    Kidney International

  2. Gentamicin treatment induces simultaneous mesangial proliferation and apoptosis in rats

    Kidney International, Vol. 65, Núm. 6, pp. 2161-2171

2003

  1. Effect of ham protein substitution on oxidative stress in older adults

    Journal of Nutrition, Health and Aging, Vol. 7, Núm. 2, pp. 84-89

2002

  1. Cyclosporin effect on rat aorta α1-adrenoceptors and their transduction mechanisms

    Journal of Cardiovascular Pharmacology, Vol. 40, Núm. 2, pp. 181-188